Care Path Log for JMD
based on the framework of Dr. Jack Kruse

"your daughter disease has made her the most accurate deuterium spectroscope man has created."

This page documents the adjunctive framework we are using with Dr. Jack Kruse alongside standard care for JMD’s FCHO1/IMD76, focusing on light, magnetism, deuterium handling, hydration and mechanical “vibration” protocols. Within that framework, deuterium‑depleted water (DDW) has been the central intervention, with the onset of DDW closely followed by rapid and unexpected improvements in both T‑cell counts and liver biochemistry, including normalization of ALT and AST over only a few months. The documents below highlight several of the more significant explanations and forum exchanges from Dr. Kruse that outline his deuterium‑centered model and how these adjunctive protocols appear to correlate with JMD’s objective clinical and laboratory outcomes.

• Dr Kruse Post explaining mechanism of IMD76

  • Extended post in which Dr. Kruse reinterprets FCHO1 deficiency/IMD76 as a “quantum communications blackout” between neuroectoderm and immune system, caused by deuterium effects at Chromosome 19. He argues that JMD’s response to DDW and environment/light changes shows that deuterium management can partially “override” this defect, and explains why he believes this adjunctive approach may be disease‑modifying despite conventional expectations.

• Jack Kruse JMD Custom Protocol

  • Written summary of Dr. Kruse’s custom protocol for JMD, explaining why he shifts from ligand/receptor pharmacology toward mechanical strain, light, and deuterium management to bypass the “seized” FCHO1/clathrin switch. He outlines how clathrin‑independent endocytosis, vitamin D signaling, cholesterol sulfation, and Chromosome 19–to–Chromosome 2 links are being targeted to restore immune and neuro‑immune function in this FCHO1 deficiency.

• Observatons: The Importance of Hydration, Food Selection and much more

  • Series of observations linking mild dehydration to acute emotional lability in JMD and showing rapid reversal after DDW and protein intake, framed as validation of a “water-over-ATP” hierarchy and the role of structured water in maintaining thalamic 40 Hz coherence. Additional notes cover her strong preference for meat/fish over sugar and limited screen interest, interpreted as her system “self‑selecting” lower‑deuterium inputs and avoiding further mitochondrial stress.

• Why Claritin works

  • Brief note and forum exchange exploring why loratadine (Claritin) consistently helps JMD’s comfort and symptoms, framed as “muting a downstream histamine noise channel” in the context of deuterium load and stagnant “exhaust.” Claritin is interpreted as a downstream symptomatic aid, while DDW plus vibration are positioned as upstream interventions on proton/exhaust physics.

• THE HUMAN GPS SYSTEM DEFINES HOW SYSTEMS OPERATE IN HUMANS

  • Dr. Kruse’s long‑form framework (“Human GPS”) describing a three‑axis (X/Y/Z) neuro‑cardiac‑immune system for managing deuterium, melanin, and mitochondrial function, with FCHO1 as a key optical‑immune bridge. This document provides the conceptual basis for many of his recommendations (light, magnetism, pressure, vagus‑focused interventions) and frames FCHO1‑related disease as a failure of this quantum “GPS” and deuterium‑shielding system.

• The Human GPS and Astaxanthin

  • Our explanatory memo on astaxanthin’s mitochondrial and gut effects, and Dr. Kruse’s detailed response situating astaxanthin as a membrane‑spanning “structural rebar” and “dielectric sealant” that supports FCHO1‑deficient membranes and vagal signaling. He describes synergy between DDW (lower deuterium load) and astaxanthin (stabilizing gut and mitochondrial membranes) in reducing inflammatory “noise” and improving appetite and gut–brain signaling.

• Observation 6.1 ALT-AST lower & Albumin

  • Case observation showing that JMD’s liver echogenic focus resolved and ALT/AST normalized for the first time, temporally associated with DDW, added astaxanthin, and 40 Hz vibration therapy during a low‑UV Toronto winter. Dr. Kruse interprets this as evidence that deuterium depletion and environmental changes are driving liver improvement, with low albumin reframed as a marker of inflammatory and electromagnetic stress rather than low protein intake.

• NAD+ NADH ratio as bio-markers for Deuterium Depletion and Lattice Coherence

  • This short post explains why Dr. Kruse views the NAD⁺/NADH ratio as a key biomarker for deuterium depletion and “lattice coherence” in the mitochondrial water network. He argues that higher NAD⁺ (and thus a higher NAD⁺/NADH ratio), together with rising albumin, indicates efficient exclusion of deuterium, good vortex/ETC function, and a “light” internal environment, whereas a shift toward NADH reflects deuterium loading, magnetic decline, and stalled mitochondrial machinery.